delangelab rockefeller.edu

The Rockefeller University Laboratory for Cell Biology and Genetics

Saturday, March 24, 2018. LABORATORY OF CELL BIOLOGY AND GENETICS. Laboratory for Cell Biology and Genetics. A TIN2 dyskeratosis congenita mutation causes telomerase-independent telomere shortening in mice. Genes Dev. 28 153-166. Super-Resolution Fluorescence Imaging of Telomeres Reveals TRF2-Dependent T-Loop Formation. A TRF1-controlled common fragile site containing interstitial telomeric sequence. S Chromosoma 121 465. Removal of shelterin reveals the telomere end-protection problem. Recent expansio.

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Galeria Aniela, the worlds local fine art gallery and Sculpture Park

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The Rockefeller University Center for Basic and Translational Research on Disorders of the Digestive System

Tuesday, April 11, 2017. Center for Basic and Translational Research on Disorders of the Digestive System. The Center for Basic and Translational Research on Disorders of the Digestive System is supported through the generosity of the Leona M. Center for Basic and Translational Research on Disorders of the Digestive System.

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The Rockefeller University Laboratory for Cell Biology and Genetics

DESCRIPTION

Saturday, March 24, 2018. LABORATORY OF CELL BIOLOGY AND GENETICS. Laboratory for Cell Biology and Genetics. A TIN2 dyskeratosis congenita mutation causes telomerase-independent telomere shortening in mice. Genes Dev. 28 153-166. Super-Resolution Fluorescence Imaging of Telomeres Reveals TRF2-Dependent T-Loop Formation. A TRF1-controlled common fragile site containing interstitial telomeric sequence. S Chromosoma 121 465. Removal of shelterin reveals the telomere end-protection problem. Recent expansio.

CONTENT

This web page had the following on the site, "Saturday, March 24, 2018." We saw that the web page said " LABORATORY OF CELL BIOLOGY AND GENETICS." It also said " Laboratory for Cell Biology and Genetics. A TIN2 dyskeratosis congenita mutation causes telomerase-independent telomere shortening in mice. Super-Resolution Fluorescence Imaging of Telomeres Reveals TRF2-Dependent T-Loop Formation. A TRF1-controlled common fragile site containing interstitial telomeric sequence. Removal of shelterin reveals the telomere end-protection problem."

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